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MPO — TLR4

Text-mined interactions from Literome

Ji et al., Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi 2008 : Ang II upregulates TLR4 expressions, and induces MPO secretion via TLR4 in RAW264.7 cells, which mediate the pro-inflammatory effect of Ang II to contribute formation and development of atherosclerosis
Ji et al., Nan Fang Yi Ke Da Xue Xue Bao 2009 : Rosiglitazone downregulated AngII induced mRNA and protein expressions of TLR4 , and inhibited MPO activity in RAW264.7 cells in a concentration dependent manner ... Rosiglitazone downregulates Ang II-induced TLR4 expression in RAW264.7 cells and inhibits MPO secretion possibly by interfering with TLR4 to relieve the inflammatory reaction, which may be one of its anti-atherosclerotic mechanisms
Chen et al., J Biomed Sci 2010 (Burns) : Commensal depletion decreased TLR4 expression as well as NF-kappaB activation of intestine, myeloperoxidase (MPO) activity as well as TNFalpha expression of lung, and bacterial killing activity of peritoneal cells
Holle et al., Rheumatology (Oxford) 2013 (Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis) : Stimulation of TLR4 and TLR9 induced MPO release, stimulation with TLR2, TLR4 and TLR9 ligands elicited IL-8 production and stimulation of TLR2 and TLR9 led to an upregulation in mPR3 expression on PMNs with no significant differences between GPA and HC after 1 or 24 h stimulation