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Text-mined interactions from Literome

Greene et al., J Biol Chem 2003 : Further, insulin stimulated c-Jun phosphorylation was not blocked by inhibitors of the phosphatidylinositol 3-kinase and mammalian target of rapamycin , indicating that c-Jun N-terminal kinase is unlikely to be the kinase phosphorylating IRS-1 Ser ( 312 ) in response to insulin
Ahmed et al., Molecular cancer 2010 (Breast Neoplasms) : Moreover, overexpression of mTOR inhibits OPN induced NF-kappaB and AP-1-DNA binding and transcriptional activity
Zheng et al., Biochem Biophys Res Commun 2012 (Urinary Bladder Neoplasms) : Further, activation of AMPK by UA promoted c-Jun N-terminal kinase (JNK) activation, but inhibited mTOR complex 1 (mTORC1) signaling to cause survivin down-regulation